Page 36 - Read Online
P. 36
Zierke et al. Rare Dis Orphan Drugs J 2023;2:10 https://dx.doi.org/10.20517/rdodj.2022.17 Page 9 of 10
Pancreatology 2022;22:1099-111. DOI
32. El Jellas K, Dušátková P, Haldorsen IS, et al. Two new mutations in the CEL gene causing diabetes and hereditary pancreatitis: how to
correctly identify MODY8 cases. J Clin Endocrinol Metab 2022;107:e1455-e1466. DOI PubMed PMC
33. Witt H, Beer S, Rosendahl J, et al. Variants in CPA1 are strongly associated with early onset chronic pancreatitis. Nat Genet
2013;45:1216-20. DOI PubMed PMC
34. Moore PC, Cortez JT, Chamberlain CE, et al. Elastase 3B mutation links to familial pancreatitis with diabetes and pancreatic
adenocarcinoma. J Clin Invest 2019;129:4676-81. DOI PubMed PMC
35. SARLES H, SARLES JC, MURATORE R, GUIEN C. Chronic inflammatory sclerosis of the pancreas-an autonomous pancreatic
disease? Am J Dig Dis 1961;6:688-98. DOI PubMed
36. Yoshida K, Toki F, Takeuchi T, Watanabe S, Shiratori K, Hayashi N. Chronic pancreatitis caused by an autoimmune abnormality.
Proposal of the concept of autoimmune pancreatitis. Dig Dis Sci 1995;40:1561-8. DOI PubMed
37. Schneider A, Michaely H, Weiss C, et al. Prevalence and incidence of autoimmune pancreatitis in the population living in the
southwest of Germany. Digestion 2017;96:187-98. DOI
38. Kanno A, Masamune A, Okazaki K, et al; Research Committee of Intractable Diseases of the Pancreas. Nationwide epidemiological
survey of autoimmune pancreatitis in Japan in 2011. Pancreas 2015;44:535-9. DOI
39. Kamisawa T, Egawa N, Inokuma S, et al. Pancreatic endocrine and exocrine function and salivary gland function in autoimmune
pancreatitis before and after steroid therapy. Pancreas 2003;27:235-8. DOI
40. Kamisawa T, Chari ST, Giday SA, et al. Clinical profile of autoimmune pancreatitis and its histological subtypes: an international
multicenter survey. Pancreas 2011;40:809-14. DOI
41. Sandrasegaran K, Menias CO. Imaging in autoimmune pancreatitis and immunoglobulin g4-related disease of the abdomen.
Gastroenterol Clin North Am 2018;47:603-19. DOI PubMed
42. Hart PA, Kamisawa T, Brugge WR, et al. Long-term outcomes of autoimmune pancreatitis: a multicentre, international analysis. Gut
2013;62:1771-6. DOI PubMed PMC
43. John DS, Aschenbach J, Krüger B, et al. Deficiency of cathepsin C ameliorates severity of acute pancreatitis by reduction of neutrophil
elastase activation and cleavage of E-cadherin. J Biol Chem 2019;294:697-707. DOI PubMed PMC
44. Ito T, Nakamura T, Fujimori N, et al. Characteristics of pancreatic diabetes in patients with autoimmune pancreatitis. J Dig Dis
2011;12:210-6. DOI
45. Zamboni G, Lüttges J, Capelli P, et al. Histopathological features of diagnostic and clinical relevance in autoimmune pancreatitis: a
study on 53 resection specimens and 9 biopsy specimens. Virchows Arch 2004;445:552-63. DOI
46. Shinagare S, Shinagare AB, Deshpande V. Autoimmune pancreatitis: a guide for the histopathologist. Semin Diagn Pathol
2012;29:197-204. DOI PubMed
47. Ikeura T, Manfredi R, Zamboni G, et al. Application of international consensus diagnostic criteria to an Italian series of autoimmune
pancreatitis. United European Gastroenterol J 2013;1:276-84. DOI PubMed PMC
48. Löhr JM, Faissner R, Koczan D, et al. Autoantibodies against the exocrine pancreas in autoimmune pancreatitis: gene and protein
expression profiling and immunoassays identify pancreatic enzymes as a major target of the inflammatory process. Am J Gastroenterol
2010;105:2060-71. DOI PubMed PMC
49. Frulloni L, Lunardi C, Simone R, et al. Identification of a novel antibody associated with autoimmune pancreatitis. N Engl J Med
2009;361:2135-42. DOI
50. Mayerle J, Sendler M, Hegyi E, Beyer G, Lerch MM, Sahin-Tóth M. Genetics, cell biology, and pathophysiology of pancreatitis.
Gastroenterology 2019;156:1951-1968.e1. DOI PubMed PMC
51. Halangk W, Lerch MM, Brandt-Nedelev B, et al. Role of cathepsin B in intracellular trypsinogen activation and the onset of acute
pancreatitis. J Clin Invest 2000;106:773-81. DOI PubMed PMC
52. Saluja A, Hashimoto S, Saluja M, Powers RE, Meldolesi J, Steer ML. Subcellular redistribution of lysosomal enzymes during
caerulein-induced pancreatitis. Am J Physiol 1987;253:G508-16. DOI PubMed
53. Ohmuraya M, Yamamura K. Roles of serine protease inhibitor Kazal type 1 (SPINK1) in pancreatic diseases. Exp Anim 2011;60:433-
44. DOI PubMed
54. Wartmann T, Mayerle J, Kähne T, et al. Cathepsin L inactivates human trypsinogen, whereas cathepsin L-deletion reduces the severity
of pancreatitis in mice. Gastroenterology 2010;138:726-37. DOI PubMed PMC
55. Aghdassi AA, John DS, Sendler M, et al. Cathepsin D regulates cathepsin B activation and disease severity predominantly in
inflammatory cells during experimental pancreatitis. J Biol Chem 2018;293:1018-29. DOI PubMed PMC
56. Gukovsky I, Gukovskaya AS, Blinman TA, Zaninovic V, Pandol SJ. Early NF-kappaB activation is associated with hormone-induced
pancreatitis. Am J Physiol 1998;275:G1402-14. DOI PubMed
57. Sendler M, Weiss FU, Golchert J, et al. Cathepsin B-mediated activation of trypsinogen in endocytosing macrophages increases
severity of pancreatitis in mice. Gastroenterology 2018;154:704-718.e10. DOI PubMed PMC
58. Gukovskaya AS, Vaquero E, Zaninovic V, et al. Neutrophils and NADPH oxidase mediate intrapancreatic trypsin activation in murine
experimental acute pancreatitis. Gastroenterology 2002;122:974-84. DOI
59. Abdulla A, Awla D, Thorlacius H, Regnér S. Role of neutrophils in the activation of trypsinogen in severe acute pancreatitis. J Leukoc
Biol 2011;90:975-82. DOI PubMed
60. Awla D, Abdulla A, Syk I, Jeppsson B, Regnér S, Thorlacius H. Neutrophil-derived matrix metalloproteinase-9 is a potent activator of