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Reiss et al. Vessel Plus 2020;4:19 I http://dx.doi.org/10.20517/2574-1209.2020.04 Page 7 of 10

Researched data for the article, discussed its content, and edited the manuscript: Voloshyna I, Glass DS,
Glass AD

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All authors declared that there are no conflicts of interest.

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REFERENCES
1. Dalen JE, Alpert JS, Goldberg RJ, Weinstein RS. The epidemic of the 20(th) century: coronary heart disease. Am J Med 2014;127:807-2.
2. Wong ND. Epidemiological studies of CHD and the evolution of preventive cardiology. Nat Rev Cardiol 2014;11:276-89.
3. Benjamin EJ, Virani SS, Callaway CW, Chamberlain AM, Chang AR, et al. Heart disease and stroke statistics-2018 update: a report from
the American Heart Association. Circulation 2018;137:e67-492.
4. Whitlock G, Lewington S, Sherliker P, Clarke R, Emberson J, et al. Body mass index and cause-specific mortality in 900,000 adults:
collaborative analyses of 57 prospective studies. Lancet 2009;373:1083-96.
5. Phillips CM. Metabolically healthy obesity: personalised and public health implications. Trends Endocrinol Metabol 2016;27:189-91.
6. Bradshaw PT, Monda KL, Stevens J. Metabolic syndrome in healthy obese, overweight, and normal weight individuals: the
Atherosclerosis Risk in Communities Study. Obesity (Silver Spring) 2013;21:203-9.
7. Fasshauer M, Blüher M. Adipokines in health and disease. Trends Pharmacol Sci 2015;36:461-70.
8. Kershaw EE, Flier JS. Adipose tissue as an endocrine organ. J Clin Endocrinol Metab 2004;89:2548-56.
9. Coelho M, Oliveira T, Fernandes R. Biochemistry of adipose tissue: an endocrine organ. Arch Med Sci 2013;9:191-200.
10. Gao X, Salomon C, Freeman DJ. Extracellular vesicles from adipose tissue - a potential role in obesity and type 2 diabetes? Front
Endocrinol (Lausanne) 2017;8:202.
11. Vieira-Potter VJ. Inflammation and macrophage modulation in adipose tissues. Cell Microbiol 2014;16:1484-92.
12. Pellegrinelli V, Carobbio S, Vidal-Puig A. Adipose tissue plasticity: how fat depots respond differently to pathophysiological cues.
Diabetologia 2016;59:1075-88.
13. Hansson GK, Libby P. The immune response in atherosclerosis: a double-edged sword. Nat Rev Immunol 2006;6:508-19.
14. Mathieu P, Poirier P, Pibarot, Lemieux I, Despres JP. Visceral obesity: the link among inflammation, hypertension, and cardiovascular
disease. Hypertension 2009;53:577-84.
15. Bobryshev YV. Monocyte recruitment and foam cell formation in atherosclerosis. Micron 2006;37:208-22.
16. Gimbrone MA Jr, García-Cardeña G. Endothelial cell dysfunction and the pathobiology of atherosclerosis. Circ Res 2016;118:620-36.
17. Bhatt A, Rohatgi A. HDL cholesterol efflux capacity: cardiovascular risk factor and potential therapeutic target. Curr Atheroscler Rep
2016;18:2.
18. Maguire EM, Pearce SWA, Xiao Q. Foam cell formation: a new target for fighting atherosclerosis and cardiovascular disease. Vascul
Pharmacol 2019;112:54-71.
19. Ley K, Miller YI, Hedrick CC. Monocyte and macrophage dynamics during atherogenesis. Arterioscler Thromb Vasc Biol 2011;31:1506-
16.
20. Feig JE, Vengrenyuk Y, Reiser V, Wu C, Statnikov A, et al. Regression of atherosclerosis is characterized by broad changes in the plaque
macrophage transcriptome. PLoS One 2012;7:e39790.
21. Moore, KJ, Sheedy FJ, Fisher EA. Macrophages in atherosclerosis: a dynamic balance. Nat Rev Immunol 2013;13:709-21.
22. Reiss AB, Anwar K, Merrill JT, Chan ES, Awadallah NW, et al. Plasma from systemic lupus patients compromises cholesterol
homeostasis: a potential mechanism linking autoimmunity to atherosclerotic cardiovascular disease. Rheumatol Int 2010;30:591-8.