Page 213 - Read Online
P. 213
Alonso-Peña et al. Cancer Drug Resist 2019;2:680-709 I http://dx.doi.org/10.20517/cdr.2019.006 Page 709
from clinical evidence to regulatory networks. J Hepatol 2017;66:424-41.
200. Araki K, Shimura T, Suzuki H, Tsutsumi S, Wada W, et al. E/N-cadherin switch mediates cancer progression via TGF-beta-induced
epithelial-to-mesenchymal transition in extrahepatic cholangiocarcinoma. Br J Cancer 2011;105:1885-93.
201. Sheng L, Zhang S, Xu H. Effect of slug-mediated down-regulation of e-cadherin on invasiveness and metastasis of anaplastic thyroid
cancer cells. Med Sci Monit 2017;23:138-43.
202. Endo K, Ashida K, Miyake N, Terada T. E-cadherin gene mutations in human intrahepatic cholangiocarcinoma. J Pathol 2001;193:310-7.
203. Lee S, Kim WH, Jung HY, Yang MH, Kang GH. Aberrant CpG island methylation of multiple genes in intrahepatic cholangiocarcinoma.
Am J Pathol 2002;161:1015-22.
204. Yang B, House MG, Guo M, Herman JG, Clark DP. Promoter methylation profiles of tumor suppressor genes in intrahepatic and
extrahepatic cholangiocarcinoma. Mod Pathol 2005;18:412-20.
205. Yamada D, Kobayashi S, Wada H, Kawamoto K, Marubashi S, et al. Role of crosstalk between interleukin-6 and transforming growth
factor-beta 1 in epithelial-mesenchymal transition and chemoresistance in biliary tract cancer. Eur J Cancer 2013;49:1725-40.
206. Zhang Y, Zeng S, Ma J, Deng G, Qu Y, et al. Nestin overexpression in hepatocellular carcinoma associates with epithelial-mesenchymal
transition and chemoresistance. J Exp Clin Cancer Res 2016;35:111.
207. Warzecha CC, Jiang P, Amirikian K, Dittmar KA, Lu H, et al. An ESRP-regulated splicing programme is abrogated during the epithelial-
mesenchymal transition. EMBO J 2010;29:3286-300.
208. Brown RL, Reinke LM, Damerow MS, Perez D, Chodosh LA, et al. CD44 splice isoform switching in human and mouse epithelium is
essential for epithelial-mesenchymal transition and breast cancer progression. J Clin Invest 2011;121:1064-74.
209. Shapiro IM, Cheng AW, Flytzanis NC, Balsamo M, Condeelis JS, et al. An EMT-driven alternative splicing program occurs in human
breast cancer and modulates cellular phenotype. PLoS Genet 2011;7:e1002218.